Ditemukan 1 dokumen yang sesuai dengan query
Farid Rahimi, editor
Amyloid-forming proteins are implicated in over 30 human diseases. The proteins involved in each disease have unrelated sequences and dissimilar native structures, but they all undergo conformational alterations to form fibrillar polymers. The fibrillar assemblies accumulate progressively into disease-specific lesions in vivo. Substantial evidence suggests these lesions are the end...
Dordrecht: [, Springer], 2012
e20417827
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