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"Summary:* In-depth discussion of effective clinical management of oral wound healing * Detailed discussion on processes such as inflammation, reepithelialization and angiogenesis * Written by experts in the field "

"Summary:Practical Periodontal Diagnosis and Treatment Planning offers its readers a step-by-step guide to diagnosing and planning treatment for periodontal patients through the latest evidence-based"

"The dynamic of Zinc (Zn) concentration was determined in saliva and blood serum related to periodontal treatment need of student Faculty of Dentistry. The Zn concentration was determined by using atomic absorption spectrometer, and questionnaire was used to measure stress condition and CPITN index was examined by using the standard hand instruments and WHO probe. Almost of concentration of Zn in saliva and blood serum were decrease significantly (p<0,001 & p<0.0S)) in stress condition. While CPITN index was increase significantly in stress condition (p<0.000I). These results suggest that Zn is linked to oral saliva ecosystem under physiological stimuli, and than Zn accumulates in the salivary gland during saliva enzyme activities. This condition supported the occur of periodontal disease."

"Telah diteliti secara klinis dan laboratoris terhadap 22 orang pasien penderita SAR dengan tujuan untuk mengetahui riwayat klinisnya dan kadar zinc dalam darah pasien pasien tersebut.Hasilnya:Dari 22 orang yang diteliti, terdapat 16 orang laki laki usia antara 16 - 77 tahun dan 6 orang wanita antara usia 19-34 tahun. Yang termasuk SAR minor 18 orang, SAR mayor 3 orang, dan SAR tipe herpes 1 orang. Berdasarkan jumlah lesi yang timbul pada satu saat serangan, 59,90% dengan lesi kurang dari 5, dan 40,10% dengan lesi lebih dari 5. Frekwensi kekembuhan dalam 1 bulan sekali 4 orang, sebulan 2 kali, 10 orang, sebulan 3 kali 2 orang, dan lebih dari 3 kali sebulan ada 6 orang.Lamanya kesembuhan lesi berkisar dari 3 hari sampai lebih dari 14 hari. Tetapi yang terbanyak, lebih dari 7 hari.Yang kemungkinan ada hubungannya dengan faktor keturunan ada 7 orang, berdasarkan anamnesa adanya anggota keluarga lainnya yang juga sering mengalami SAR.Lamanya SAR berlangsung berkisar antara 3 hari sampai lebih dari 2 minggu.Pada umumnya antara 7-10 hari. Hasil pemeriksaan kadar Zinc dalam darah, yang terendah adalah 0,karena terlalu rendah sehingga tidak terdeteksi oleh cara AAS.Yang tertinggi adalah 9,4000 ppm. Terdapat 18 orang (82 %) yang kadar zinc dalam darahnya dibawah normal."

"ABSTRAKOrofacial pain includes pain associated with the hard and soft tissues of the head, face, neck and all of the intraoral structures. Upon painful episodes, consuming analgesics or traditional medicine are relatively common. It is also a common sense that high cholesterol and hypertension may contribute to the pain. Since most pain sufferers are women, estrogen is proposed to be a modulator of pain perception. Nevertheless, the mechanism of pain modulation in women is still in controversy. Systemic manivestations of periodontal disease are widely accepted. However, the role of pedodontal disease as an etiology of orofacial and musculoskeletal pains is rarely discussed. Recent study in medical psychoneuroimmunology may reveal the possibility of periodontal disease as an etiology of these painful symptoms. The objective of this study is to reveal the possibility of periodontal disease as an etiology of orofacial and musculoskeletal pains. Periodontal treatments were done to women who suffered from orofacial and musculoskeletal pains, resulting in the dissapearing of the symptoms. Regarding to the amazing results, the conclusion is that especially in women, periodontal disease may cause orofacial and musculoskeletal pains especially in women."

"ABSTRACTPeriodontal disease has been defined as the pathological processes that involve the periodontium. These pathological changes may be limited to one of the constituent parts of the periodontium as in chronic gingivitis, or include several or all of the tissues. Most periodontal diseases are specific to the periodontium, but in some cases they may be manifestations of general disease or diseases of other organs WHO, TRS 207, 1961). Such a definition or description of periodontal disease is somewhat all encompassing. Indeed, some indices developed to measure periodontal disease appear to have considered the condition as a continuum of a single disease entity. Thus, the periodontal index (Russell 1956) uses an increasing scoring system as gingival inflammation worsens, pocketing develops and tooth mobility occurs. Similarly the Periodontal Disease Index (Ramfjord 1959) scores higher as gingivitis develops and loss of attachment occurs. In our present state of knowledge it would appear that periodontal disease is not a single entity with different degrees of severity. The umbrella title periodontal disease encompasses a number of disease states, some of which may be specific infections in a susceptible host. Unfortunately, our knowledge is still incomplete so that the full picture has yet to be unraveled.A variety of classifications for periodontal disease have been proposed with broad agreement for the terms chronic gingivitis, chronic adult periodontitis, prepubertal periodontitis, chronic juvenile periodontitis, and rapidly progressive (adult) periodontitis (Gotlieb 1923, Butler 1969, Baer 1971, Page et al 1983a,b, Bystrom et al 1983, Cutress 1986). Further subdivisions of these disease states have been suggested, and include descriptive terminology such as localized and generalized (Baer 1971, Page et al 1983a). Additionally, the condition refractory periodontitis has been suggested to describe disease, which has failed to respond to conventional therapy (Gordon et al 1985).Despite the identification of apparently distinct chronic disease states of the periodontal tissues, it would appear most of the conditions diagnosed by present day clinical techniques fall within the categories of chronic gingivitis or chronic (adult) periodontitis. Perhaps fortunately, the other disease entities are rare (Page et al 1983a,b, Bystrom et al 1983, Cutress 1986).The one factor common to all the chronic disease states of the periodontal tissues appears to be a bacterial aetiology or at least a bacterial association. As with all "infections" host factors play an important role in the susceptibility and progress of disease. Nevertheless, bacterial deposits are always found in association with periodontal disease states and at present prevention and treatment is aimed at elimination of bacteria from tissues sites. The association of bacterial plaque to periodontal disease was first derived from epidemiological data (Ash et al 1964). Evidence for a direct aetiological role of bacterial plaque, at least in the initiation of chronic gingivitis was shown by subsequent clinical experimentation (Lae et al 1965).The specificity of the bacteria in plaque to disease states is still hotly debated. Universal acceptance of one of either the non-specific plaque hypothesis (Miller 1890) or the specific plaque hypothesis (Tanner et al 1979) has not occurred.Even for chronic gingivitis it appears uncertain whether clinically measurable inflammation occurs in association with a critical plaque mass or a significant increase in the proportions of certain bacterial species for example, Actinomyces species, Fusobacterium nucleatum, Bacteroides rnelaninogenicus and Bacteroides intermedius (Loesche and Syed 1978, Moore et al 1982a, Moore et al 1987).Similarly in chronic adult periodontitis, there is strong association between Gram-negative anaerobic bacteria and disease (Slots 1977b, Spiegel et al 1979, Tanner et al 1984, Mandell et al 1987). Furthermore certain organisms are frequently reported at sites of disease activity or rapid disease progression and include Bacteroides gingivalis, Fusobacterium nucleatum, Bacteroides forsythus, Elkenella corrodens, Bacteroides intermedius and Actinobacillus actinomycetemcomitans (Tanner et al 1979, 1984). However, these data are derived, for the most part, from extremely small numbers of patients. Moreover, it must be remember that in excess of 300 species and 35 genera of bacteria have been identified from human periodontal sites (Moore 1987). A further 100 or more species are thought to exist, but have as yet proved impossible to isolate and identify.To date there have been no epidemiological studies of the microflora associated with periodontal disease. In fact, the feasibility of microbiological testing in the field has not even been assessed. The nearest attempts to large-scale oral microbiology surveys have been from clinical trials of oral hygiene products looking for adverse shifts in flora or resistance to antimicrobial agents (Volpe et al 1969a, Stallard et al 1969, Schlott et al 1976)."

"Periodontal disease is one of the risk factors for certain systemic conditions. Retrospective and prospective studies showed that pregnant women with periodontitis have significantly higher risk to deliver preterm low birth weight infants (gestation period < 37 weeks, and birth weight < 2500 grams) and the risk increase as the periodontitis progressed during pregnancy. Periodontal disease could mediate this systemic effect through the action of periodontal pathogens and lipopolysaccharide increasing the production of prostaglandin and cytokine (physiologic mediators in parturition) which will trigger the delivery of preterm low birth weight infants. Periodontal therapy significantly reduces the rates of preterm low birth weight among women with periodontitis. It is concluded that periodontal disease is an independent risk factor for preterm low birth weight, and inclusion of oral prophylaxis procedure during pregnancy should be considered."

"Latar belakang: Populasi di Asia memiliki beberapa faktor risiko periodontitis terkaitanatomi dan mikroorganisme dalam rongga mulutnya. Periodontitis merupakan ancamanbesar terhadap kesehatan mulut dan dapat menimbulkan gejala perubahan klinis sepertimunculnya tanda-tanda inflamasi serta terjadinya peningkatan pocket probing depth(PPD) dan clinical attachment loss (CAL) yang dapat menyebabkan rasa tidak nyamanpada penderitanya baik dalam aspek fisik, psikologis, maupun sosial. Tujuan: Untukmenganalisis pengaruh terapi periodontal terhadap nilai OHRQoL pada penderitaperiodontitis di Asia dari studi yang menggunakan kuesioner OHIP-14. Metode: Ujimeta-analisis serta penyusunan systematic review (PROSPERO CRD42020203254)dengan pencarian literatur pada online database yaitu PubMed, Scopus, dan EBSCO.Studi yang diidentifikasi kemudian melalui tahapan skrining, penilaian eligibilitas, daninklusi menggunakan pedoman Preferred Reporting Items for Systematic Reviews andMeta-Analyses (PRISMA). Hasil: Sebanyak enam studi memenuhi kriteria inklusi untukdilakukan systematic review dan empat studi dengan intervensi terapi periodontal nonbedahdiikutsertakan dalam meta-analisis. Analisis kuantitatif dilakukan pada tigarentang waktu follow-up yaitu minggu ke-1 dan 2 dengan mean difference [95% CI]: -13,31 [-33,71 ; 7,10], minggu ke-4 dan 5 dengan mean difference [95% CI]: -16,12 [-35,27 ; 3,03], serta minggu ke 9 hingga 12 dengan mean difference [95% CI]: -4,14 [-6,85 ; -1,43]. Kesimpulan: Terapi periodontal dapat meningkatkan OHRQoL penderitaperiodontitis di Asia. Peningkatan tersebut dapat terlihat paling signifikan pada mingguke-4 dan 5 pasca terapi.

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Background: Asians have periodontitis risk factors regarding to the anatomy andmicroorganisms found in their oral cavity. Periodontitis is one of the most prevalentdiseases that affects the oral cavity, causing several symptoms such as inflammation andincrease in pocket probing depth (PPD) and clinical attachment loss (CAL). Symptomscaused by periodontitis may cause discomfort in some aspects of life such as physical,psychological, and social aspect. Objective: To analyze the impacts of periodontaltherapy on OHRQoL in periodontitis patients in Asia from studies using OHIP-14questionnaire. Methods: Meta-analysis and systematic review (PROSPEROCRD42020203254) of the studies obtained from three databases (PubMed, Scopus, andEBSCO). Identified studies were screened and assessed following the PreferredReporting Items for Systematic Reviews and Meta-Analyses (PRISMA) guidelines.Results: From 641 studies retrieved, six met the criteria for qualitative analysis. Studiesusing non-surgical periodontal treatment are also included for meta-analysis. Quantitativeanalysis were conducted by categorizing the follow-up period into three groups: 1-2weeks follow-up with mean difference [95% CI]: -13.31 [-33.71 ; 7.10], 4-5 weeksfollow-up with mean difference [95% CI]: -16.12 [-35.27 ; 3.03], and 9-12 weeks followupwith mean difference [95% CI]: -4.14 [-6.85 ; -1.43]. Conclusion: Periodontal therapycan enhance the OHRQoL of periodontitis patients in Asia. The most significant impactcan be seen on the follow-up period of 4-5 weeks"