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Cellular responses to stress including DNA damage show multiple options involving the mechanisms of growth arrest, DNA repair and programmed cell death or apoptosis. Failures in these mechanisms can result in oncogenesis or accelerated senescence. Much of the response is coordinated by p53, a nuclear phosphoprotein with a central role...
Jakarta: Journal of Dentistry Indonesia, 2003
AJ-Pdf
Artikel Jurnal Universitas Indonesia Library
